Author name: Kristen Hartman

Brown HD, Kozak R, Sarter M (2006) Bilateral removal of cholinergic inputs to the medial prefrontal cortex disrupts the ability of rats to cope with challenges on attentional performance. Neuroscience 2006 Abstracts 369.20. Society for Neuroscience, Atlanta, GA. Summary: Studies using microdialysis for the measurement of the release of neurotransmitters in task-performing animals demonstrated attentional […]

Newman LA (2006) Comparison of the effects of selective cholinergic or noradrenergic deafferentation in the medial, prefrontal cortex on sustained attention. Neuroscience 2006 Abstracts 369.21. Society for Neuroscience, Atlanta, GA. Summary: Acetylcholine (ACH) and norepinephrine (NE) have been shown to be critically important in controlling the activity of cortical neurons during attention demanding tasks. ACH

Pasumarthi RK, Fadel J (2006) Anatomical and neurochemical mediators of nicotine-induced activation of orexin neurons. Neuroscience 2006 Abstracts 369.22. Society for Neuroscience, Atlanta, GA. Summary: Orexin/hypocretin neurons of the lateral hypothalamus and contiguous perifornical area (LH/PFA) are important for state-dependent behavior and metabolic regulation. These neurons are activated-as indicated by Fos expression-by a variety of

Frederick-Duus D, Fadel J (2006) Orexin transmission is required for food-related increases in cortical acetylcholine release. Neuroscience 2006 Abstracts 369.23. Society for Neuroscience, Atlanta, GA. Summary: The hypothalamic orexin/hypocretin neuropeptides (OxA and OxB) are crucial modulators of state-dependent behavior including the regulation of arousal in response to homeostatic challenges. Orexins provide a moderately dense innervation

Broussard JI, Venugopal S, Sarter M, Givens B (2006) Cholinergic modulation of posterior parietal neuronal activity associated with the detection of signals in attentional task-performing rats. Neuroscience 2006 Abstracts 369.7. Society for Neuroscience, Atlanta, GA. Summary: The posterior parietal cortex (PPC) is considered a major component of the brains’ attention systems, specifically of the orientation

Likhtik E, Apergis-Schoute J, Pare D (2006) A selective lesioning method to probe the role of intercalated (ITC) amygdala neurons in the extinction of classically conditioned fear responses. Neuroscience 2006 Abstracts 370.19. Society for Neuroscience, Atlanta, GA. Summary: The acquisition of conditioned fear responses (CRs) is thought to involve the potentiation of synapses conveying information

Iacobucci P, Colonnello V, Ricceri L (2006) Detailed analysis of ultrasound vocalizations emitted by 12-d-old rats during homing test: effects of a brief reunion with littermates in neonatal basal forebrain cholinergic lesioned pups. Neuroscience 2006 Abstracts 374.17. Society for Neuroscience, Atlanta, GA. Summary: To evaluate the role of the developing cholinergic basal forebrain system on

McKay LC, Feldman JL (2006) Unilateral ablation of neurokinin-1 receptor-expressing (NK1R) neurons within the preBötzinger complex (preBötC) in adult rats disrupts breathing during sleep but not during wakefulness. Neuroscience 2006 Abstracts 455.4. Society for Neuroscience, Atlanta, GA. Summary: In adult rats, as the number of ablated preBötC NK1R neurons increases, breathing becomes increasingly disrupted during

Li A, Nattie EE (2006) Lesions of neurokinin-1 receptor immunoreactive (NK1R-ir) neurons in the ventral medulla decrease chemoreception and ventilation. Neuroscience 2006 Abstracts 455.5. Society for Neuroscience, Atlanta, GA. Summary: We injected SSP-saporin, a toxin specific for NK1R-ir neurons, into the cisterna magna in order to lesion a wide aspect of the ventral medulla. NK1R-ir

Shiromani PJ, Blanco-Centurion C, Xu M, Murillo-Rodriguez E, Gerashchenko D, Hof PR (2006) Adenosine and sleep debt in the basal forebrain. Neuroscience 2006 Abstracts 458.13. Society for Neuroscience, Atlanta, GA. Summary: The waxing and waning of the sleep drive is hypothesized to be regulated by endogenous sleep factors acting on specific neurons in the brain.