Author name: Kristen Hartman

The role of the cholinergic basal forebrain in learning, memory and reward expectancies.

Savage LM, Buzzette R, Ramirez D (2001) The role of the cholinergic basal forebrain in learning, memory and reward expectancies. Neuroscience 2001 Abstracts 314.18. Society for Neuroscience, San Diego, CA. Summary: The cholinergic basal forebrain degenerates in Alzheimer’s Disease and the degree of this degeneration correlates with a decline in cognitive processing. In the present […]

The role of the cholinergic basal forebrain in learning, memory and reward expectancies. Read More »

Nicotine enhances N-methyl-D-aspartate receptor responses and facilitates long-term potentiation in the hippocampus from rats with cholinergic lesions.

Hamaue N, Yamazaki Y, Ohmori H, Sumikawa K (2001) Nicotine enhances N-methyl-D-aspartate receptor responses and facilitates long-term potentiation in the hippocampus from rats with cholinergic lesions. Neuroscience 2001 Abstracts 376.5. Society for Neuroscience, San Diego, CA. Summary: Nicotine reverses cognitive impairments caused by lesion of the cholinergic system and improves performance of Alzheimer’s patients. The

Nicotine enhances N-methyl-D-aspartate receptor responses and facilitates long-term potentiation in the hippocampus from rats with cholinergic lesions. Read More »

Maze strategy in rats with GABAergic or cholinergic lesions of medial septum.

Yoder RM, Reuss SA, Pang KC (2001) Maze strategy in rats with GABAergic or cholinergic lesions of medial septum. Neuroscience 2001 Abstracts 418.7. Society for Neuroscience, San Diego, CA. Summary: Cholinergic and GABAergic neurons are the two major cell types that project from medial septum to hippocampus. Although complete lesions of hippocampus or medial septum

Maze strategy in rats with GABAergic or cholinergic lesions of medial septum. Read More »

Selective neonatal neurochemical lesions persist into old age and cause Alzheimer-like pathology.

de Butte M, Fortin T, Sherren N, Pappas BA (2001) Selective neonatal neurochemical lesions persist into old age and cause Alzheimer-like pathology. Neuroscience 2001 Abstracts 426.8. Society for Neuroscience, San Diego, CA. Summary: There is a strong negative correlation between forebrain acetylcholine (ACH) function and the depth of Alzheimer’s dementia (AD). Forebrain norepinephrine (NE) is

Selective neonatal neurochemical lesions persist into old age and cause Alzheimer-like pathology. Read More »

A novel method for localized sympathetic nervous system denervation of peripheral tissue using guanethidine.

Bartness TJ, Demas GE (2001) A novel method for localized sympathetic nervous system denervation of peripheral tissue using guanethidine. Neuroscience 2001 Abstracts 309.10. Society for Neuroscience, San Diego, CA. Summary: A simple technique for the functional deactivation of the sympathetic nervous system innervation of peripheral tissues is described using the local application of guanethidine. Multiple

A novel method for localized sympathetic nervous system denervation of peripheral tissue using guanethidine. Read More »

Noradrenergic inputs to the paraventricular nucleus (PVN) is involved in estrogen receptor α expression in the PVN of 48-h fasted and 2DG-injected female rats.

Estacio MA, Tsukamura H, Reyes BA, Maeda KI (2001) Noradrenergic inputs to the paraventricular nucleus (PVN) is involved in estrogen receptor α expression in the PVN of 48-h fasted and 2DG-injected female rats. Neuroscience 2001 Abstracts 409.6. Society for Neuroscience, San Diego, CA. Summary: Involvement of noradrenergic inputs to the paraventricular nucleus (PVN) in estrogen

Noradrenergic inputs to the paraventricular nucleus (PVN) is involved in estrogen receptor α expression in the PVN of 48-h fasted and 2DG-injected female rats. Read More »

Central origin of ataxic breathing after lesion of preBötzinger complex (preBötc) neurokinin 1 receptor expressing (NK1R+) neurons.

Janczewski WA, Gray PA, Feldman JL (2001) Central origin of ataxic breathing after lesion of preBötzinger complex (preBötc) neurokinin 1 receptor expressing (NK1R+) neurons. Neuroscience 2001 Abstracts 243.2. Society for Neuroscience, San Diego, CA. Summary: Pathological breathing results from near complete lesions of preBötC NK1R+ neurons in awake adult rats (Gray et al., FASEB J.,15,

Central origin of ataxic breathing after lesion of preBötzinger complex (preBötc) neurokinin 1 receptor expressing (NK1R+) neurons. Read More »

Dose-dependent effects of intrathecal substance P-saporin and SSP-saporin.

Wiley RG, Kline IV RH, Lappi DA (2001) Dose-dependent effects of intrathecal substance P-saporin and SSP-saporin. Neuroscience 2001 Abstracts 281.11. Society for Neuroscience, San Diego, CA. Summary: Selective destruction of lamina I dorsal horn neurons expressing the neurokinin-1 receptor (NK-1R) can attenuate responses to capsaicin injection and thermal hyperalgesia/mechanical allodynia in models of inflammatory, persistent

Dose-dependent effects of intrathecal substance P-saporin and SSP-saporin. Read More »

Hypocretin-saporin (Hcrt-sap) as a tool to examine hypocretin function.

Gerashchenko D, Kohls MD, Greco MA, Waleh NS, Salin-Pascual R, Kilduff TS, Lappi DA, Shiromani PJ (2001) Hypocretin-saporin (Hcrt-sap) as a tool to examine hypocretin function. Neuroscience 2001 Abstracts 410.7. Society for Neuroscience, San Diego, CA. Summary: The hypocretin (Hcrt) peptides are linked to narcolepsy. Humans with narcolepsy have decreased numbers of Hcrt neurons and

Hypocretin-saporin (Hcrt-sap) as a tool to examine hypocretin function. Read More »

Light- and food-entrained circadian rhythms in rats with neurotoxic lesions of hypocretin cells.

Mistlberger RE, Antle MC, Jones M, Kilduff TS (2001) Light- and food-entrained circadian rhythms in rats with neurotoxic lesions of hypocretin cells. Neuroscience 2001 Abstracts 410.8. Society for Neuroscience, San Diego, CA. Summary: The hypocretins (Hcrt1 & 2) are lateral hypothalamic (LH) neuropeptides implicated in the regulation of feeding and sleep-wake states. Hcrt lesions attenuate

Light- and food-entrained circadian rhythms in rats with neurotoxic lesions of hypocretin cells. Read More »

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