Author name: Kristen Hartman

Effects of N-desmethylclozapine on attentional performance following loss of basal forebrain corticopetal cholinergic inputs.

Maness EBL, Leong CS, Burk JA (2016) Effects of N-desmethylclozapine on attentional performance following loss of basal forebrain corticopetal cholinergic inputs. Neuroscience 2016 Abstracts 833.15 / HHH34. Society for Neuroscience, San Diego, CA. Summary: Corticopetal cholinergic neurons play a vital role in attentional processing, and dysregulation of this system contributes to central nervous system disorders […]

Effects of N-desmethylclozapine on attentional performance following loss of basal forebrain corticopetal cholinergic inputs. Read More »

Preventing falls in PD in a rat model of impaired cognitive control of complex movements by a pro-cholinergic combination treatment.

Kucinski AJ, De Jong IEM, Sarter M (2016) Preventing falls in PD in a rat model of impaired cognitive control of complex movements by a pro-cholinergic combination treatment. Neuroscience 2016 Abstracts 835.15 / III34. Society for Neuroscience, San Diego, CA. Summary: Parkinson’s disease (PD) patients, in addition to primary motor symptoms resulting from extensive losses

Preventing falls in PD in a rat model of impaired cognitive control of complex movements by a pro-cholinergic combination treatment. Read More »

Developmental disturbances in thalamocortical connection are sufficient to produce almost all features of schizophrenia.

Rajakumar R (2016) Developmental disturbances in thalamocortical connection are sufficient to produce almost all features of schizophrenia. Neuroscience 2016 Abstracts 845.01 / LLL41. Society for Neuroscience, San Diego, CA. Summary: Partial ablation of subplate of the developing frontal cortex was achieved by two different approaches in PD1 S-D rat pups: infusions of either P75 receptor

Developmental disturbances in thalamocortical connection are sufficient to produce almost all features of schizophrenia. Read More »

Cerebral cholinergic mechanisms in pain: CBF lesions vs systemic scopolamine.

Wiley RG, Yezierski R, Vierck Jr CJ (2016) Cerebral cholinergic mechanisms in pain: CBF lesions vs systemic scopolamine. Neuroscience 2016 Abstracts 525.15 / SS2. Society for Neuroscience, San Diego, CA. Summary: Cholinergic inputs to the cerebral cortex and limbic system, originating primarily from the cholinergic basal forebrain (CBF), play an important role in cortical sensory

Cerebral cholinergic mechanisms in pain: CBF lesions vs systemic scopolamine. Read More »

Maternal aggression is impaired by prepartum serotonin-specific lesions of the midbrain dorsal raphe.

Vitale EM, Holschbach MA, Lonstein JS (2016) Maternal aggression is impaired by prepartum serotonin-specific lesions of the midbrain dorsal raphe. Neuroscience 2016 Abstracts 338.01 / SS14. Society for Neuroscience, San Diego, CA. Summary: The postpartum period in laboratory rats and other animals is characterized by increased maternal responsiveness, decreased anxiety, and increased aggression. Pharmacologically manipulating

Maternal aggression is impaired by prepartum serotonin-specific lesions of the midbrain dorsal raphe. Read More »

A unique subdivision of serotonergic neurons in the dorsal raphe nucleus projects to the basolateral amygdala complex to enhance fear-conditioned behaviors.

Bernabe CS, Caliman IF, Abreu ARR, Shekhar A, Johnson PL (2016) A unique subdivision of serotonergic neurons in the dorsal raphe nucleus projects to the basolateral amygdala complex to enhance fear-conditioned behaviors. Neuroscience 2016 Abstracts 74.23 / GGG14. Society for Neuroscience, San Diego, CA. Summary: The basolateral and lateral amygdala nuclei complex (BLC) is implicated

A unique subdivision of serotonergic neurons in the dorsal raphe nucleus projects to the basolateral amygdala complex to enhance fear-conditioned behaviors. Read More »

Striatal cholinergic interneurons: their depletion and its progression.

Abudukeyoumu N, Garcia-Munoz M, Jaidar OP, Arbuthnott G (2016) Striatal cholinergic interneurons: their depletion and its progression. Neuroscience 2016 Abstracts 245.09 / RR4. Society for Neuroscience, San Diego, CA. Summary: Even before the discovery that Parkinson’s was produced by the loss of dopaminergic neurons, this neurological disease was treated with anticholinergic drugs. A balance between

Striatal cholinergic interneurons: their depletion and its progression. Read More »

Basal forebrain cholinergic neurons are vital for sleepiness observed after alcohol consumption.

Sharma A, Sharma R, Sahota P, Thakkar M (2016) Basal forebrain cholinergic neurons are vital for sleepiness observed after alcohol consumption. Neuroscience 2016 Abstracts 254.12 / AAA18. Society for Neuroscience, San Diego, CA. Summary: Purpose: Corticopetal wake-promoting basal forebrain region (BF) is implicated to mediate sleepiness following alcohol intake. However, the specific phenotype of the

Basal forebrain cholinergic neurons are vital for sleepiness observed after alcohol consumption. Read More »

Catecholaminergic innervation and the neuronal activation of hypothalamic glucose sensitive regions during rapid- and slow-onset hypoglycemia in adult male rats..

Jokiaho A, Watts AG (2016) Catecholaminergic innervation and the neuronal activation of hypothalamic glucose sensitive regions during rapid- and slow-onset hypoglycemia in adult male rats.. Neuroscience 2016 Abstracts 256.21 / CCC20. Society for Neuroscience, San Diego, CA. Summary: Hypoglycemic counterregulation is mediated by glucosensors located in the hypothalamus, hindbrain, and portal-mesenteric veins. But which are

Catecholaminergic innervation and the neuronal activation of hypothalamic glucose sensitive regions during rapid- and slow-onset hypoglycemia in adult male rats.. Read More »

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