Author name: Kristen Hartman

Lack of effect of moderate Purkinje cell loss on working memory.

Wrenn CC, Wiley RG (2001) Lack of effect of moderate Purkinje cell loss on working memory. Neuroscience 107(3):433-445. doi: 10.1016/s0306-4522(01)00326-8 Summary: When 192-Saporin (Cat. #IT-01) is injected intracerebroventricularly, some p75-expressing cerebellar Purkinje cells are eliminated along with cholinergic neurons. To verify that the effects of basal forebrain lesions on working memory were not caused by […]

Lack of effect of moderate Purkinje cell loss on working memory. Read More »

Novel method for localized, functional sympathetic nervous system denervation of peripheral tissue using guanethidine.

Demas GE, Bartness TJ (2001) Novel method for localized, functional sympathetic nervous system denervation of peripheral tissue using guanethidine. J Neurosci Methods 112:21-28. doi: 10.1016/s0165-0270(01)00452-6 Summary: Sympathectomy, or surgical interruption of sympathetic nerve pathways, is an important technique in the analysis of the sympathetic nervous system. The authors investigate and compare several different methods of

Novel method for localized, functional sympathetic nervous system denervation of peripheral tissue using guanethidine. Read More »

Selective immunotoxin lesion of spinally projecting norepineprhine and epinephrine (NE/E) neurons impairs the glucagon response to 2-deoxy-d-glucose (2DG).

Dinh TT, Sanders NM, Pedrow C, Ritter S (2001) Selective immunotoxin lesion of spinally projecting norepineprhine and epinephrine (NE/E) neurons impairs the glucagon response to 2-deoxy-d-glucose (2DG). Neuroscience 2001 Abstracts 947.1. Society for Neuroscience, San Diego, CA. Summary: Previous work has shown that the targeted immunotoxin, anti-dopamine ß-hydroxylase conjugated to saporin (DSAP), can be used

Selective immunotoxin lesion of spinally projecting norepineprhine and epinephrine (NE/E) neurons impairs the glucagon response to 2-deoxy-d-glucose (2DG). Read More »

Immunotoxin lesion of catecholaminergic neurons innervating the medial hypothalamus elevates basal expression of and attenuates glucoprivaation-induced increases in agouti gene related protein (AGRP) mRNA.

Fraley GS, Dinh TT, Ritter S (2001) Immunotoxin lesion of catecholaminergic neurons innervating the medial hypothalamus elevates basal expression of and attenuates glucoprivaation-induced increases in agouti gene related protein (AGRP) mRNA. Neuroscience 2001 Abstracts 947.2. Society for Neuroscience, San Diego, CA. Summary: Catecholaminergic (CA) innervation of medial hypothalamic structures is necessary for glucoprivation-induced feeding, glucocorticoid

Immunotoxin lesion of catecholaminergic neurons innervating the medial hypothalamus elevates basal expression of and attenuates glucoprivaation-induced increases in agouti gene related protein (AGRP) mRNA. Read More »

Selective immunotoxin lesion of hypothalamically-projecting norepinephrine/epinephrine (NE/E) neurons impairs the glucocorticoid response to glucoprivation.

Ritter S, Dinh TT, Sanders NM, Pedrow C (2001) Selective immunotoxin lesion of hypothalamically-projecting norepinephrine/epinephrine (NE/E) neurons impairs the glucocorticoid response to glucoprivation. Neuroscience 2001 Abstracts 947.3. Society for Neuroscience, San Diego, CA. Summary: Decreased glucose utilization triggers behavioral and neuroendocrine responses that increase blood glucose concentrations and delivery of glucose to the brain. These

Selective immunotoxin lesion of hypothalamically-projecting norepinephrine/epinephrine (NE/E) neurons impairs the glucocorticoid response to glucoprivation. Read More »

Targeted lesions of Substance P receptor cells in L3-4 lumbar spinal cord severely impair male ejaculatory behavior.

Truitt WA, Stanton LA, Coolen LM (2001) Targeted lesions of Substance P receptor cells in L3-4 lumbar spinal cord severely impair male ejaculatory behavior. Neuroscience 2001 Abstracts 958.18. Society for Neuroscience, San Diego, CA. Summary: Previously we demonstrated the existence of a spinothalamic pathway in the male rat where neural activation is specifically induced by

Targeted lesions of Substance P receptor cells in L3-4 lumbar spinal cord severely impair male ejaculatory behavior. Read More »

Hyperphagia and obesity results from the injection of the immunotoxin neuropeptide Y (NPY)-saporin (NPY-sap) into the paraventricular hypothalamus (PVH) of rats.

Bugarith K, Ritter S, Dinh T (2001) Hyperphagia and obesity results from the injection of the immunotoxin neuropeptide Y (NPY)-saporin (NPY-sap) into the paraventricular hypothalamus (PVH) of rats. Neuroscience 2001 Abstracts 948.2. Society for Neuroscience, San Diego, CA. Summary: NPY is a peptide implicated in the control of numerous physiological processes. A variety of G-protein

Hyperphagia and obesity results from the injection of the immunotoxin neuropeptide Y (NPY)-saporin (NPY-sap) into the paraventricular hypothalamus (PVH) of rats. Read More »

A monoclonal antibody to an extracellular domain of the serotonin transporter: Characterization and targeting properties.

Kohls MD, Majer KA, Russell BJ, Han Q, Blakely RD, Lappi DA (2001) A monoclonal antibody to an extracellular domain of the serotonin transporter: Characterization and targeting properties. Neuroscience 2001 Abstracts 814.9. Society for Neuroscience, San Diego, CA. PMID: 0 Summary: Using a peptide corresponding to a sequence from an extracellular domain of the rat

A monoclonal antibody to an extracellular domain of the serotonin transporter: Characterization and targeting properties. Read More »

Bcl-2 improves survival of lesioned cholinergic neurons.

Lee KY, Leos R, Borowski TB, de Lacalle S (2001) Bcl-2 improves survival of lesioned cholinergic neurons. Neuroscience 2001 Abstracts 803.6. Society for Neuroscience, San Diego, CA. Summary: Alzheimer’s disease is associated with the progressive cell death of cholinergic neurons in the central nervous system (CNS). A current challenge is to develop therapeutic approaches that

Bcl-2 improves survival of lesioned cholinergic neurons. Read More »

Monoamine modulation of spinal reflex excitability of the lower limb in the rat: Intrathecal infusion (i.t.) of anti-DBH saporin toxin – time course for behavior.

Bose P, Wang DC, Parmer R, Wiley RG, Thompson FJ (2001) Monoamine modulation of spinal reflex excitability of the lower limb in the rat: Intrathecal infusion (i.t.) of anti-DBH saporin toxin – time course for behavior. Neuroscience 2001 Abstracts 771.3. Society for Neuroscience, San Diego, CA. Summary: Progressive neurophysiological changes in the excitability of ankle

Monoamine modulation of spinal reflex excitability of the lower limb in the rat: Intrathecal infusion (i.t.) of anti-DBH saporin toxin – time course for behavior. Read More »

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