Author name: Kristen Hartman

Depletion of IB4-binding sensory neurons results in elevated nociceptive thresholds.

Vulchanova L, Stone LS, Olson T, Riedl MS, Elde R, Honda CN (1999) Depletion of IB4-binding sensory neurons results in elevated nociceptive thresholds. Neuroscience 1999 Abstracts 272.8. Society for Neuroscience, Miami, FL. Summary: The lectin IB4 binds to and is specifically taken up by a subset of small sensory neurons, proposed to play a role […]

Depletion of IB4-binding sensory neurons results in elevated nociceptive thresholds. Read More »

NGF-mediated alteration of NF-κB binding activity after partial immuno-lesions to rat cholinergic basal forebrain neurons.

Gu Z, Toliver-Kinsky T, Glasgow J, Cain L, Perez-Polo JR (1999) NGF-mediated alteration of NF-κB binding activity after partial immuno-lesions to rat cholinergic basal forebrain neurons. Neuroscience 1999 Abstracts 300.15. Society for Neuroscience, Miami, FL. Summary: Memory loss and cognitive deficits in die aged and in patients with Alzheimer’s disease (AD) are often associated with

NGF-mediated alteration of NF-κB binding activity after partial immuno-lesions to rat cholinergic basal forebrain neurons. Read More »

Lesions of basal forebrain cholinergic neurons eliminate the modulatory effects of benzodiazepine receptor ligands on behavioral and cardiovascular reactions in an anxiogenic paradigm.

Stowell JR, Bemtson GG, Sarter M (1999) Lesions of basal forebrain cholinergic neurons eliminate the modulatory effects of benzodiazepine receptor ligands on behavioral and cardiovascular reactions in an anxiogenic paradigm. Neuroscience 1999 Abstracts 358.5. Society for Neuroscience, Miami, FL. Summary: We have previously shown that basal forebrain cortical cholinergic projections mediate potentiation of the cardiovascular

Lesions of basal forebrain cholinergic neurons eliminate the modulatory effects of benzodiazepine receptor ligands on behavioral and cardiovascular reactions in an anxiogenic paradigm. Read More »

Exciting New Advances in Pain Research and Treatment Using Receptor Internalization Technologies – SFN 1999 Symposium

Meetings Abstracts /

Symposium:  “Exciting New Advances in Pain Research and Treatment Using Receptor Internalization Technologies” Society for Neuroscience (SFN) Symposium October 23-28, 1999 – Miami, Florida D.A. Thomas, NIDA-NIH (co-chair); R.G. Wiley, Vanderbilt University; W.J. Martin, UCSF; M.J. Iadorola, NIDCR-NIH; P. Mantyh, University of Minnesota; R.M. Caudle, University of Florida (co-chair)  A press conference was held prior

Exciting New Advances in Pain Research and Treatment Using Receptor Internalization Technologies – SFN 1999 Symposium Read More »

SFN Poster of the Year 1999

Meetings Abstracts /

Awarded by ATS at Society for Neuroscience (SFN) Miami, Florida • October 23-28, 1999 474.6 Abolition of cyanide-induced sympathoexcitation by selective lesion of bulbospinal catecholaminergic neurons. A.M. SCHREIHOFER* and P.G. GUYENET Univ. of Virginia. Cardiovascular regulation: central control III (POSTER) The rostral ventrolateral medulla (RVLM) contains BS C1 adrenergic cells and non-CA neurons whose relative

SFN Poster of the Year 1999 Read More »

Abolition of cyanide-induced sympathoexcitation by selective lesion of bulbospinal catecholaminergic neurons

Schreihofer AM, Guyenet PG (1999) Abolition of cyanide-induced sympathoexcitation by selective lesion of bulbospinal catecholaminergic neurons. Neuroscience 1999 Abstracts 474.6. Society for Neuroscience, MIami, FL. Summary: The rostral ventrolateral medulla (RVLM) contains BS C1 adrenergic cells and non-CA neurons whose relative importance for the production of sympathetic vasomotor tone and cardiovascular reflexes remains unknown. In

Abolition of cyanide-induced sympathoexcitation by selective lesion of bulbospinal catecholaminergic neurons Read More »

Cognitive changes and modified processing of amyloid precursor protein in the cortical and hippocampal system after cholinergic synapse loss and muscarinic receptor activation.

Lin L, Georgievska B, Mattsson A, Isacson O (1999) Cognitive changes and modified processing of amyloid precursor protein in the cortical and hippocampal system after cholinergic synapse loss and muscarinic receptor activation. Proc Natl Acad Sci U S A 96:12108-12113. doi: 10.1073/pnas.96.21.12108 Related Products: 192-IgG-SAP (Cat. #IT-01)

Cognitive changes and modified processing of amyloid precursor protein in the cortical and hippocampal system after cholinergic synapse loss and muscarinic receptor activation. Read More »

Lesions of the C1 catecholaminergic neurons of the ventrolateral medulla in rats using anti-DBH-saporin.

Madden CJ, Ito S, Rinaman L, Wiley RG, Sved AF (1999) Lesions of the C1 catecholaminergic neurons of the ventrolateral medulla in rats using anti-DBH-saporin. Am J Physiol 46:R1063-R1075. doi: 10.1152/ajpregu.1999.277.4.R1063 Summary: The authors review the use of anti-DBH-SAP (Cat. #IT-03) to study the role of C1 neurons within the rostral ventromedial medulla in cardiovascular

Lesions of the C1 catecholaminergic neurons of the ventrolateral medulla in rats using anti-DBH-saporin. Read More »

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