Author name: Kristen Hartman

Kline IV RH, Wiley RG (2009) Spinal µ-opiate receptor (MOR)-expressing dorsal horn neurons: Role in modulating pain and opiate analgesia. Neuroscience 2009 Abstracts 560.13/CC72. Society for Neuroscience, Chicago, IL. Summary: Selective destruction of MOR-expressing interneurons in lamina II of the dorsal horn of the spinal cord increases reflex nocifensive responses to formalin and decreases the […]

Tai S, Leung L (2009) Facilitation of dentate gyrus population spike may involve septohippocampal GABAergic input. Neuroscience 2009 Abstracts 566.18/EE33. Society for Neuroscience, Chicago, IL. Summary: The medial septum (MS) is known to modulate the neural circuitry in the hippocampus. It have been demonstrated that, in anaesthetized rats, MS stimulation prior to perforant path stimulation

Gaykema RP, Thacker GC, Shapiro NJ, Goehler LE (2009) Immunotoxic lesion of hypothalamic noradrenergic/adrenergic input ameliorates the effects of peripheral LPS challenge on sickness behavior and associated brain c-Fos expression. Neuroscience 2009 Abstracts 570.11/EE120. Society for Neuroscience, Chicago, IL. Summary: Caudal medullary catecholamine neurons that innervate the hypothalamus play a major role in the activation

Huang T-Y, Lin L-S, Chen H-I, Jen C (2009) Chronic treadmill exercise improves cerebellar functions: Alterations in mitochondrial protein expression, rotarod performance, and toxin resistance. Neuroscience 2009 Abstracts 660.18/CC34. Society for Neuroscience, Chicago, IL. Summary: The effects of exercise on cerebellar functions were studied. Five-week-old male Wistar rats were divided into exercise and sedentary groups.

Thomsen MS, Hay-Schmidt A, Hansen HH, Mikkelsen JD (2009) Poster: Distinct neural pathways mediate alpha7 nicotinic acetylcholine receptor-dependent activation of the forebrain. Neuroscience 2009 Abstracts 646.2/V14. Society for Neuroscience, Chicago, IL. Summary: α7 nicotinic acetylcholine receptor (nAChR) agonists are novel drugs candidates for the treatment of cognitive deficits in schizophrenia, which have shown pro-cognitive effects

Laplante FP, Dufresne M, Lappi DA, Sullivan RM (2009) Depletion of cholinergic neurons in the nucleus accumbens impairs dopamine function in the prefrontal cortex in the rat. Neuroscience 2009 Abstracts 341.7/O16. Society for Neuroscience, Chicago, IL. Summary: Studies of post mortem schizophrenic brains have revealed a selective loss of cholinergic interneurons, most pronounced in the

Brink TS, Khasabov SG, Fliss PM, Simone DA (2009) Ablation of NK-1 expressing neurons in the rostral ventromedial medulla attenuates inflammatory hyperalgesia. Neuroscience 2009 Abstracts 361.5/BB31. Society for Neuroscience, Chicago, IL. Summary: Substance P (SP) is a neuropeptide synthesized by many nociceptive primary sensory neurons and is released into the spinal cord following noxious stimulation

Li A-J, Wang Q, Dinh TT, Ritter S (2009) Leptin-saporin injection into the arcuate nucleus lesions NPY/AGRP and POMC neurons and produces hyperphagia, obesity and changes in diurnal feeding patterns in rats. Neuroscience 2009 Abstracts 374.5/EE116. Society for Neuroscience, Chicago, IL. Summary: Leptin-saporin (Lep-SAP), a conjugate of leptin with a ribosomal inactivating toxin, saporin (Advanced

Carvalho AF, Reyes AS, Van Bockstaele EJ (2009) The role of limbic norepinephrine in cannabinoid-induced aversion. Neuroscience 2009 Abstracts 449.3/V29. Society for Neuroscience, Chicago, IL. Summary: The endocannabinoid system has been implicated in diverse physiological mechanisms including modulation of pain and analgesia, learning and memory and feeding, among others. Thus, targeting the cannabinoid system has

Freiria-Oliveira AH, Blanch GT, De Paula PM, Colombari E, Menani JV, Colombari DS (2009) Role of A2 noradrenergic neurons and angiotensinergic mechanisms on hypotension induced by hemorrhage. Neuroscience 2009 Abstracts 467.18/DD70. Society for Neuroscience, Chicago, IL. Summary: The A2 catecholaminergic neurons in the commissural subdivision of the nucleus tractus solitarii (cNTS) are activated by hemorrhage.