Somatostatin Receptor-3 (SSTR3) Peptide [PR-06]

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Somatostatin Receptor-3 is one of five receptor subtypes termed SSTR1-5. They are G-protein-coupled receptors characterized by seven transmembrane domains with an extracellular amino terminal domain and an intracellular carboxy terminus. These receptors function in the regulation of numerous physiological processes such as the secretion of insulin, glucagon, and growth hormone, as well as cell growth […]

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Somatostatin Receptor-4 (SSTR4) Peptide [PR-07]

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Somatostatin Receptor-4 is one of five receptor subtypes termed SSTR1-5. They are G-protein-coupled receptors characterized by seven transmembrane domains with an extracellular amino terminal domain and an intracellular carboxy terminus. These receptors function in the regulation of numerous physiological processes such as the secretion of insulin, glucagon, and growth hormone, as well as cell growth

Somatostatin Receptor-4 (SSTR4) Peptide [PR-07] Read More »

Somatostatin Receptor-5 (SSTR5) Peptide [PR-08]

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Somatostatin Receptor-5 is one of five receptor subtypes termed SSTR1-5. They are G-protein-coupled receptors characterized by seven transmembrane domains with an extracellular amino terminal domain and an intracellular carboxy terminus. These receptors function in the regulation of numerous physiological processes such as the secretion of insulin, glucagon, and growth hormone, as well as cell growth

Somatostatin Receptor-5 (SSTR5) Peptide [PR-08] Read More »

Reducing Agent in Media

Q:  We will be using your chick-ZAP secondary conjugate (Cat. #IT-62) and noticed that in your protocol you mention not to use a reducing agent in your media. Our normal growth media contains beta mercaptoethanol at 100 μM. Will this be a problem? A: Officially, we would recommend allowing the cells to acclimate to media

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A common neuronal mechanism of hypertension and sleep disturbances in spontaneously hypertensive rats: Role of orexinergic neurons.

Cui S-Y, Huang Y-L, Cui X-Y, Zhao H-L, Hu X, Liu Y-T, Qin Y, Kurban N, Zhang Y-H (2020) A common neuronal mechanism of hypertension and sleep disturbances in spontaneously hypertensive rats: Role of orexinergic neurons. Prog Neuropsychopharmacol Biol Psychiatry 100:109902. doi: 10.1016/j.pnpbp.2020.109902 Objective: To investigate dynamic changes in sleep patterns during the development of

A common neuronal mechanism of hypertension and sleep disturbances in spontaneously hypertensive rats: Role of orexinergic neurons. Read More »

Participation of brainstem monoaminergic nuclei in behavioral depression.

Lin Y, Sarfraz Y, Jensen A, Dunn AJ, Stone EA (2011) Participation of brainstem monoaminergic nuclei in behavioral depression. Pharmacol Biochem Behav 100(2):330-9. doi: 10.1016/j.pbb.2011.08.021 Summary: While the classical model states reductions in central noradrenergic activity produces depression, more recent work has indicated that higher activity in this brain region directly correlates with depression. Using

Participation of brainstem monoaminergic nuclei in behavioral depression. Read More »

Neural stem cells and cholinergic neurons: Regulation by immunolesion and treatment with mitogens, retinoic acid, and nerve growth factor.

Calza L, Giuliani A, Fernandez M, Pirondi S, D’Intino G, Aloe L, Giardino L (2003) Neural stem cells and cholinergic neurons: Regulation by immunolesion and treatment with mitogens, retinoic acid, and nerve growth factor. Proc Natl Acad Sci U S A 100(12):7325-7330. doi: 10.1073/pnas.1132092100 Summary: The authors explore the influence of exogenous administration of hormones,

Neural stem cells and cholinergic neurons: Regulation by immunolesion and treatment with mitogens, retinoic acid, and nerve growth factor. Read More »

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Effective Date: 09-Aug-2023Last Updated: 09-Aug-2023 What are cookies? This Cookie Policy explains what cookies are and how we use them, the types of cookies we use i.e, the information we collect using cookies and how that information is used, and how to manage the cookie settings. Cookies are small text files that are used to

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Lack of capsaicin (CAP)-evoked sensitization following SP-SAP treatment is not attributed to decreased CAP-evoked exciataion

Khasabov SG, Rogers SD, Ghilardi JR, Peters CM, Mantyh PW, Simone DA (2002) Lack of capsaicin (CAP)-evoked sensitization following SP-SAP treatment is not attributed to decreased CAP-evoked exciataion. Neuroscience 2002 Abstracts 351.24. Society for Neuroscience, Orlando, FL. Summary: The depletion of SPR+ neurons in the spinal cord by substance P-saporin conjugate (SP-SAP) prevents the development

Lack of capsaicin (CAP)-evoked sensitization following SP-SAP treatment is not attributed to decreased CAP-evoked exciataion Read More »

Impaired oxygen-sensitive regulation of mitochondrial biogenesis within the von Hippel-Lindau syndrome

Li S, Li W, Yuan J, Bullova P, Wu J, Zhang X, Liu Y, Plescher M, Rodriguez J, Bedoya-Reina OC, Jannig PR, Valente-Silva P, Yu M, Henriksson MA, Zubarev RA, Smed-Sörensen A, Suzuki CK, Ruas JL, Holmberg J, Larsson C, Christofer Juhlin C, von Kriegsheim A, Cao Y, Schlisio S (2022) Impaired oxygen-sensitive regulation of

Impaired oxygen-sensitive regulation of mitochondrial biogenesis within the von Hippel-Lindau syndrome Read More »

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