Author name: Kristen Hartman

Neuromuscular plasticity in a mouse neurotoxic model of spinal motoneuronal loss.

Gulino R, Vicario N, Giunta MAS, Spoto G, Calabrese G, Vecchio M, Gulisano M, Leanza G, Parenti R (2019) Neuromuscular plasticity in a mouse neurotoxic model of spinal motoneuronal loss. Int J Mol Sci 20(6):1500. doi: 10.3390/ijms20061500 Objective: To use a neurotoxic model of spinal motoneuron depletion, induced by injection of CTB-SAP, to investigate the […]

Neuromuscular plasticity in a mouse neurotoxic model of spinal motoneuronal loss. Read More »

Waterpipe smoke effect on oxidative stress levels in buccal cells using MAWI i-SWAB tubes

Mouawad J, Azar JA, Bassil M (2019) Waterpipe smoke effect on oxidative stress levels in buccal cells using MAWI i-SWAB tubes. Biomark J 5(1):1-6. doi: 10.36648/2472-1646.5.1.57 PMID: 909090 Objective: To evaluate the effect of water pipe smoke on oxidative stress levels in buccal cells, using MAWI i-SWAB tubes for the detection of different protein oxidation

Waterpipe smoke effect on oxidative stress levels in buccal cells using MAWI i-SWAB tubes Read More »

Inflammatory mediators of opioid tolerance: Implications for dependency and addiction.

Eidson LN, Murphy AZ (2019) Inflammatory mediators of opioid tolerance: Implications for dependency and addiction. Peptides 115:51-58. doi: 10.1016/j.peptides.2019.01.003 Objective: To determine what mediates opioid tolerance and alterations in glutamate homeostasis. Summary: Site-specific lesions of PAG MOR-containing neurons using Dermorphin-SAP significantly reduce the antinociceptive effects of systemic morphine suggesting that PAG MOR is critical for

Inflammatory mediators of opioid tolerance: Implications for dependency and addiction. Read More »

Kangxianling decoction prevents renal fibrosis in rats with 5/6 nephrectomy and inhibits Ang II-induced ECM production in glomerular mesangial cells.

Ji J, Tao P, He L (2019) Kangxianling decoction prevents renal fibrosis in rats with 5/6 nephrectomy and inhibits Ang II-induced ECM production in glomerular mesangial cells. J Pharmacol Sci 139(4):367-372. doi: 10.1016/j.jphs.2019.03.003 PMID: 30929858 Objective: To investigate effects of Kangxianling decoction on renal fibrosis in a rat model of 5/6 Nephrectomy (5/6 N) in

Kangxianling decoction prevents renal fibrosis in rats with 5/6 nephrectomy and inhibits Ang II-induced ECM production in glomerular mesangial cells. Read More »

Inhibition of methamphetamine self-administration and reinstatement by central blockade of angiotensin II receptor in rats.

Xu X, Pan J, Li X, Cui Y, Mao Z, Wu B, Xu H, Zhou W, Liu Y (2019) Inhibition of methamphetamine self-administration and reinstatement by central blockade of angiotensin II receptor in rats. J Pharmacol Exp Ther 369(2):244-258. doi: 10.1124/jpet.118.255729 PMID: 30867225 Objective: To explore the role of central angiotensin II receptor (ATR) in

Inhibition of methamphetamine self-administration and reinstatement by central blockade of angiotensin II receptor in rats. Read More »

Knockdown of fidgetin improves regeneration of injured axons by a microtubule-based mechanism.

Matamoros AJ, Tom VJ, Wu D, Rao Y, Sharp DJ, Baas PW (2019) Knockdown of fidgetin improves regeneration of injured axons by a microtubule-based mechanism. J Neurosci 39(11):2011-2024. doi: 10.1523/JNEUROSCI.1888-18.2018 PMID: 30647150 Objective: To test whether fidgetin knockdown assists axonal regeneration. Summary: Results show that DRG neurons in which fidgetin was knocked down displayed enhanced

Knockdown of fidgetin improves regeneration of injured axons by a microtubule-based mechanism. Read More »

Serotonin and motherhood: From molecules to mood.

Pawluski JL, Li M, Lonstein JS (2019) Serotonin and motherhood: From molecules to mood. Front Neuroendocrinol 53:100742. doi: 10.1016/j.yfrne.2019.03.001 Summary: Serotonin may affect how mothers perceive or behaviorally readjust to changes in the sensory cues emitted by their offspring as they age. The DR serotonin-lesioned mothers studied by Holschbach and colleagues (2018) were much less

Serotonin and motherhood: From molecules to mood. Read More »

Cholinergic deficit induced by central administration of 192IgG-Saporin is associated with activation of microglia and cell loss in the dorsal hippocampus of rats

Dobryakova YV, Volobueva MN, Manolova AO, Medvedeva TM, Kvichansky AA, Gulyaeva NV, Markevich VA, Stepanichev MY, Bolshakov AP (2019) Cholinergic deficit induced by central administration of 192IgG-Saporin is associated with activation of microglia and cell loss in the dorsal hippocampus of rats. Front Neurosci 13:146. doi: 10.3389/fnins.2019.00146 Objective: To study the histopathology of the hippocampus

Cholinergic deficit induced by central administration of 192IgG-Saporin is associated with activation of microglia and cell loss in the dorsal hippocampus of rats Read More »

Spinal α2-adrenoceptors and neuropathic pain modulation; therapeutic target.

Bahari Z, Meftahi GH (2019) Spinal α2-adrenoceptors and neuropathic pain modulation; therapeutic target. Br J Pharmacol 176(14):2366-2381. doi: 10.1111/bph.14580 Objective: To provide an an overview of the cellular mechanisms through which brainstem adrenergic descending inhibitory processing can alter spinal pain transmission to the higher centres, and how these pathways change in neuropathic pain conditions focusing

Spinal α2-adrenoceptors and neuropathic pain modulation; therapeutic target. Read More »

Noradrenergic terminals are the primary source of α2-adrenoceptor mediated dopamine release in the medial prefrontal cortex.

Devoto P, Flore G, Saba P, Scheggi S, Mulas G, Gambarana C, Spiga S, Gessa GL (2019) Noradrenergic terminals are the primary source of α2-adrenoceptor mediated dopamine release in the medial prefrontal cortex. Prog Neuropsychopharmacol Biol Psychiatry 90:97-103. doi: 10.1016/j.pnpbp.2018.11.015 Objective: To clarify the relative contribution of dopamine (DA) release from noradrenergic and dopaminergic terminals

Noradrenergic terminals are the primary source of α2-adrenoceptor mediated dopamine release in the medial prefrontal cortex. Read More »

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